MKL1

Rôles

Notes et références

1. ↑ ^{a b et c} GRCh38: Ensembl release 89: ENSG00000196588 - Ensembl, May 2017
2. ↑ ^{a b et c} GRCm38: Ensembl release 89: ENSMUSG00000042292 - Ensembl, May 2017
3. ↑ « Publications PubMed pour l'Homme », sur National Center for Biotechnology Information, U.S. National Library of Medicine
4. ↑ « Publications PubMed pour la Souris », sur National Center for Biotechnology Information, U.S. National Library of Medicine
5. ↑ Wang DZ, Li S, Hockemeyer D et al. Potentiation of serum response factor activity by a family of myocardin-related transcription factors, Proc Natl Acad Sci U S A, 2002;99:14855–14860
6. ↑ Small EM, Thatcher JE, Sutherland LB et al. Myocardin-related transcription factor-a controls myofibroblast activation and fibrosis in response to myocardial infarction' , Circ Res, 2010;107:294–304
7. ↑ Kuwahara K, Kinoshita H, Kuwabara Y et al. Myocardin-related transcription factor A is a common mediator of mechanical stress- and neurohumoral stimulation-induced cardiac hypertrophic signaling leading to activation of brain natriuretic peptide gene expression,,Mol Cell Biol, 2010;30:4134–4148
8. ↑ Yang Y, Cheng X, Tian W et al. MRTF-A steers an epigenetic complex to activate endothelin-induced pro-inflammatory transcription in vascular smooth muscle cells, Nucleic Acids Res, 2014;42:10460–10472
9. ↑ Weng X, Yu L, Liang P et al. Endothelial MRTF-A mediates angiotensin II induced cardiac hypertrophy, J Mol Cell Cardiol, 2015;80:23–33
10. ↑ Yu L, Yang G, Zhang X et al. Megakaryocytic leukemia 1 bridges epigenetic activation of NADPH oxidase in macrophages to cardiac ischemia-reperfusion injury, Circulation, 2018;138:2820–2836