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Wasik, MA Show undefined attributes

created [InstanceEdit:9701293] Rothfels, Karen, 2020年09月26日

dbId 9701386

displayName Wasik, MA

firstname Mariusz A

initial MA

publications

[LiteratureReference:9701758] STAT5A is epigenetically silenced by the tyrosine kinase NPM1-ALK and acts as a tumor suppressor by reciprocally inhibiting NPM1-ALK expression
[LiteratureReference:9701290] Nucleophosmin-anaplastic lymphoma kinase: the ultimate oncogene and therapeutic target
[LiteratureReference:9846094] Nucleophosmin/anaplastic lymphoma kinase (NPM/ALK) oncoprotein induces the T regulatory cell phenotype by activating STAT3
[LiteratureReference:9846132] STAT3 induces transcription of the DNA methyltransferase 1 gene (DNMT1) in malignant T lymphocytes
[LiteratureReference:9846133] STAT3- and DNA methyltransferase 1-mediated epigenetic silencing of SHP-1 tyrosine phosphatase tumor suppressor gene in malignant T lymphocytes
[LiteratureReference:9701292] Inhibition of ALK enzymatic activity in T-cell lymphoma cells induces apoptosis and suppresses proliferation and STAT3 phosphorylation independently of Jak3
[LiteratureReference:9701412] Multilevel dysregulation of STAT3 activation in anaplastic lymphoma kinase-positive T/null-cell lymphoma
[LiteratureReference:9701294] Oncogenic kinase NPM/ALK induces through STAT3 expression of immunosuppressive protein CD274 (PD-L1, B7-H1)
[LiteratureReference:9846102] Oncogenic tyrosine kinase NPM-ALK induces expression of the growth-promoting receptor ICOS
[LiteratureReference:9846073] Malignant transformation of CD4+ T lymphocytes mediated by oncogenic kinase NPM/ALK recapitulates IL-2-induced cell signaling and gene expression reprogramming

schemaClass Person

surname Wasik

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