Reactome | Inghirami, G

created [InstanceEdit:9648462] Orlic-Milacic, Marija, 2019年06月06日

crossReference

[DatabaseIdentifier:9729894] ORCID:0000-0001-5566-0864

dbId 9648424

displayName Inghirami, G

firstname Giorgio

initial G

modified [InstanceEdit:9729893] Rothfels, Karen, 2021年05月04日

orcidId 0000-0001-5566-0864

publications

[LiteratureReference:9702103] PRDM1/BLIMP1: a tumor suppressor gene in B and T cell lymphomas
[LiteratureReference:9667881] The anaplastic lymphoma kinase in the pathogenesis of cancer
[LiteratureReference:9850777] NPM-ALK oncogenic tyrosine kinase controls T-cell identity by transcriptional regulation and epigenetic silencing in lymphoma cells
[LiteratureReference:9701321] Anaplastic lymphoma kinase (ALK) activates Stat3 and protects hematopoietic cells from cell death
[LiteratureReference:9710822] The tyrosine phosphatase Shp2 interacts with NPM-ALK and regulates anaplastic lymphoma cell growth and migration
[LiteratureReference:9701357] Stat3 is required for ALK-mediated lymphomagenesis and provides a possible therapeutic target
[LiteratureReference:9701314] The Transcriptional Regulator Sin3A Contributes to the Oncogenic Potential of STAT3
[LiteratureReference:9701343] NPM-ALK transgenic mice spontaneously develop T-cell lymphomas and plasma cell tumors
[LiteratureReference:9850784] The lymphoma-associated NPM-ALK oncogene elicits a p16INK4a/pRb-dependent tumor-suppressive pathway
[LiteratureReference:9725240] Functional validation of the anaplastic lymphoma kinase signature identifies CEBPB and BCL2A1 as critical target genes
[LiteratureReference:9846055] IRF4 Mediates the Oncogenic Effects of STAT3 in Anaplastic Large Cell Lymphomas
[LiteratureReference:9648432] Genetics of follicular lymphoma transformation
[LiteratureReference:9846040] Gene expression profiling uncovers molecular classifiers for the recognition of anaplastic large-cell lymphoma within peripheral T-cell neoplasms
[LiteratureReference:9846160] Whole Exome Sequencing reveals NOTCH1 mutations in anaplastic large cell lymphoma and points to Notch both as a key pathway and a potential therapeutic target
[LiteratureReference:9701411] PRDM1/BLIMP1 is commonly inactivated in anaplastic large T-cell lymphoma
[LiteratureReference:9845961] Convergent mutations and kinase fusions lead to oncogenic STAT3 activation in anaplastic large cell lymphoma

schemaClass Person

surname Inghirami

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