Spreading Depression, Spreading
Depolarizations, and the Cerebral Vasculature.
Mechanical stimulation of the myocardium resulting in electrical events is well described, occurring in such circumstances as catheter-induced ectopy during intracardiac procedures and in the production of premature ventricular
depolarizations by thumping the chest during asystole [13].
It explains technical aspects and general concepts of electrophysiologic investigation, sinus node function, atrioventricular conduction, intraventricular conduction disturbances, miscellaneous phenomena related to atrioventricular conduction, ectopic rhythms and premature
depolarizations, supraventricular tachycardias, atrial flutter and fibrillation, preexcitation syndromes, recurrent ventricular tachycardia, the evaluation of antiarrhythmic agents, and catheter and surgical ablation in the therapy of arrhythmias.
Action potential duration lengthening and consequently prolonged repolarization cause early after
depolarizations (EADs) due to activation or delayed inactivation of inward depolarizing sodium and L type calcium currents.
The Involvement of G Protein on SCRT-Induced
Depolarizations in Pacemaker Potentials in Cultured ICCs.
In 7 SG neurons tested in current clamp mode, taurine (100 [micro]M) induced repeated membrane
depolarizations (Figure 1(a)).
Such spreading injury
depolarizations occur repetitively over hours and days and propagate throughout the unhealthy, but not yet depolarized or necrotic tissue (e.g., ischemic penumbra).
Experimental evidence has been provided that phase 2 early after
depolarizations (EAD) can be generated from hypertrophied left ventricular wall in the absence of action potential duration (APD) prolonging agents.
Possibly first discussed as an intrinsic potential by Watanabe (1958) in lobster CG, and further described in studies of the Squilla (stomatopod) CG (Watanabe et al., 1967a, b), driver potentials are relatively slow, sustained, regenerative
depolarizations that may arise from a gradual pacemaker potential or be evoked by a
depolarization, such as an excitatory synaptic potential (or an applied depolarizing stimulus) (Fig.
Healthy, asymptomatic men whose exercise testing shows frequent premature ventricular
depolarizations are at the same increased risk for later cardiovascular death as those whose exercise tests reveal ischemia, reported Dr.